In the series, we will provide documented evidence of death and severe injuries linked with Gardasil, analyze the root cause of its harm, and offer solutions.
The True Culprits of Cervical Cancer Beyond HPV
There are major root causes for cervical cancer that are often ignored, yet their consideration is pivotal for achieving a cancer-free life.
In the series, "The HPV Vaccine: A Double-Edged Sword?" we will provide documented evidence of death and severe injuries linked with Gardasil, analyze the root cause of its harm, and offer solutions.To prevent cervical cancer, the human papillomavirus (HPV) vaccine has been extensively promoted for young girls and women to protect against high-risk HPV infections that can cause cervical cancer later in life, as an HPV infection is regarded as the main risk factor for cervical cancer.
The Vulnerable Surface of the CervixThroughout humanity, the role of a woman has been closely intertwined with marriage and childbirth, impacting her lifelong health and well-being.
Under the effect of hormones during early puberty, the transformation zone forms and gradually shifts in its location surrounding the cervix area, becoming stronger as a woman matures.
However, at a young age, this zone is not yet well developed or strong enough to defend against viruses and other harmful factors.
When girls enter puberty, a vulnerable type of cells, known as columnar cells, covers a certain part of the cervix. Only when we mature does another more protective type of cell, called the squamous epithelium, form a layer over the vulnerable columnar cells.
Thus, if sexual behavior occurs during adolescence, this zone will be much more vulnerable to being attacked by a virus, especially the HPV.
Once attacked by the HPV, the protective layers of the cervix's outer lining will be removed, exposing the underlying layer to the HPV and allowing a precancerous lesion to more easily develop. For young girls who don't yet have protective layers, it's even more risky.
A Spanish study revealed that women under the age of 25 were 39 times more likely to engage in sexual activity before the age of 18 than those over the age of 55. This indicates that unhealthy sexual behavior has become more prevalent in younger generations. Similar phenomena have been reported in Brazil.Therefore, engaging in sexual activity at a young age, having rough sex, or contracting other sexually transmitted infections, can increase the likelihood of HPV entry and infection, thereby increasing the risk of developing cervical cancer during our lifetime.
Age at First Sex MattersThe largest dataset based on a series of multinational case-control etiological studies on cervical cancer was conducted by the International Agency for Research on Cancer (IARC) in France and the Institut Català d'Oncologia in Spain.
An etiological study is a study designed to uncover the cause of a disease.
The analysis included 1,864 cases of invasive cervical carcinoma and 1,719 corresponding controls from developing countries: Morocco, Algeria, Philippines, Thailand, India, Brazil, Colombia, Paraguay, and Peru. Ninety-five percent of the cancer patients and 17 percent of the control group tested positive for HPV DNA. Of those cancer cases, a majority (92 percent), had squamous cell carcinoma.
Engaging in sex for the first time on or before age 16 or between 17 to 20 years of age increased the risk of developing invasive cervical cancer by 2.3 and 1.8 times, respectively, compared with women over the age of 21.
The results remained consistent even after the investigators adjusted the model to include the potential confounding factors of HPV infection status, age, country, lifetime number of sexual partners, parity, and education level.
Number of Sexual Partners MattersThere are three primary reasons that the number of sexual partners matters with cervical cancer.
Third, the number of sexual partners is also a significant risk factor for cervical cancer.
In 1989, researchers at Teen Colposcopy Clinic at the University of California–San Francisco published an epidemiological study of risk factors for precancerous lesions of cervical cancer based on an investigation of 14- to 19-year-old women who visited their clinic.
The group with multiple sexual partners consisted of patients who had more than one male sexual partner in the last six months. The non-multiple sexual partners group included patients with the same male partner, those who were not sexually active in the previous six months, and those who occasionally had sex with the same male partner.
The multiple sexual partners group was significantly associated with a positive HPV outcome, including HPV 16 or 18 and other high-risk types of HPV infections. They also had a significantly higher percentage of more severe precancerous lesions, particularly CIN-II and CIN-III. CIN refers to cervical intraepithelial neoplasia, the presence of abnormal cell growth (neoplasia) on the surface of the outer lining of the cervix (intraepithelial tissue). It is also known as cervical dysplasia.
Age at First Pregnancy MattersIf people tend to have sex at a younger age and have multiple sexual partners, the risk of getting pregnant at an earlier age increases.
The transformation zone of the cervix is sensitive to hormones, particularly estrogen and progesterone.
Combined Oral Contraceptives Increase Cancer RiskTo avoid an unplanned pregnancy, many women use oral contraceptives. In July 2023, the U.S. Food and Drug Administration approved the first nonprescription oral contraceptive, Opill, a progestin-only pill. Although it does not contain estrogen and is expected to be available over-the-counter in early 2024, it does come with risks and side effects.
Combined oral contraceptives (containing both estrogen and progestogen) are carcinogenic to humans; this assessment was made partly on the basis of the increased risk for cancer of the cervix.
Researchers found that the relative risk of cervical cancer significantly increased in current users of oral contraceptives and with a longer duration of use, then declined once use subsided.
For example, 10 years of using oral contraceptives started at around age 20 to 30 years is estimated to increase the cumulative incidence of invasive cervical cancer occurring by age 50 by 13 percent in less developed countries and 18 percent in more developed countries.Cervical cancer also shares several common risk factors with other cancers, including family cancer history, smoking, and a weakened immune system.
Gardasil 9 Doesn't Protect Against All High-Risk StrainsWhile the HPV vaccine is a potential way to prevent HPV infection, it could come with serious unexpected consequences.
When people receive the HPV vaccine, the viral strains not covered by the vaccine may become more prevalent and assume a more dominant role in the cervix of the vaccinated women. This means the HPV vaccine has the potential to trigger changes in vaginal microbiota. The virus has found ways to escape vaccine-induced immunity.
Uncertain Protection With HPV VaccinesDespite claims of efficacy, the exact level of protection of HPV vaccines is uncertain.
Our immune system consists of two parts: natural immunity and adaptive immunity. Natural immunity fights a wide range of pathogens like bacteria and other germs regardless of their gene codes, strains, or variants.
HPV vaccines injected into a person's muscle mainly boost adaptive immunity—more specifically, they stimulate T and B cells and antibodies (also called immunoglobulins) generated by the B cells. There are different types of immunoglobulins, depending on the place where they stay and exert their effects.
In the case of HPV, our innate or natural immunity, especially mucosal immunity, plays a major role in preventing cervical cancer.
Many people argue that the HPV vaccine offers good protection against cervical cancer and is based on solid data. However, their data is not bulletproof and is questionable.
Observational studies and registry data have shown evidence supporting the protective effects of the HPV vaccine against cervical cancer and precancerous lesions. However, it's important when interpreting these study results to consider whether data related to the major risk factors previously discussed were collected, analyzed, and balanced across the different study groups.
The study did not adequately assess the major risk factors between the vaccinated and unvaccinated groups (e.g., age at first sex or pregnancy, number of sexual partners, oral contraceptive use, smoking, or general health status).
The NEJM study suggested that the overall level of protection conferred by the HPV vaccine is estimated to be around 60 percent (and up to 88 percent for younger girls). However, as we have previously elucidated, had the main factors associated with cervical cancer risk been included and thoroughly analyzed, the study conclusions would likely have been invalidated.
Natural Immunity Remains EssentialYou may wonder what could happen if you don't get the HPV vaccine. Will you be left with no protection? Or how will you advise your children to protect themselves against cervical cancer if they do not take the HPV vaccination?
Regardless of whether or not someone receives an HPV vaccination, our natural immunity remains essential to our protection against persistent HPV.
More specifically, our natural immunity against the virus primarily resides with our mucosal immunity.
Natural immunity does not only fight against the virus, it also fights against cancer.
Our bodies are naturally equipped with a defense system that can fight cancer cells. The key lies in optimizing our natural immunity, thus enabling a strong immune system that can promptly detect and monitor any signs of cancer cells.
In our battle against cancer, it is crucial to maintain a constant focus on strengthening our immune system and exerting control over known risk factors. Adopting a holistic approach is actually the most important thing we can do—with zero side effects. Eating healthy food, maintaining quality sleep and a healthy weight, exercising, mindfulness programs, meditation, and stress-reduction techniques are all helpful to our immunity.
Our Choices MatterThere are undeniable safety risks associated with HPV vaccines. One of the most difficult questions is deciding whether these risks outweigh the benefits.
The HPV vaccine is not a golden shield protecting against cervical cancer. Its claims of protection are highly questionable and its harms are well-documented.
Let's instead wisely focus our attention on how to strengthen our immunity and avoid the high-risk behaviors with the greatest impact on our cervical health. Maintaining responsible attitudes towards sexuality is an effective and natural way to protect ourselves.
We all possess a sacred gift—our naturally endowed immunity. By controlling our risk behaviors and bolstering our natural immunity, we can confidently embrace a more holistic, dynamic, and robust approach to safeguarding ourselves against HPV infection and cervical cancer.